CHMP gives nod for Arrowhead’s Redemplo in familial chylomicronemia syndrome

Arrowhead Pharmaceuticals has received a positive opinion from the European Medicines Agency’s Committee for Medicinal Products for Human Use (CHMP) recommending authorization of Redemplo (plozasiran) as an adjunct to diet for reducing triglyceride levels in adult patients with familial chylomicronemia syndrome (FCS). If confirmed by the European Commission, which is expected to issue a decision in Q2 2026, Redemplo would become the first small interfering RNA (siRNA) medicine authorized in the EU for FCS — and notably, the first therapy in that market indicated for both genetically confirmed and clinically diagnosed patients.

The CHMP opinion follows regulatory approvals in the US, Canada, and China, extending what has been a multi-jurisdictional program for Arrowhead’s lead commercial asset. The molecule targets apolipoprotein C-III (apoC-III), a hepatically produced protein that inhibits triglyceride catabolism and clearance. By silencing the mRNA encoding apoC-III using Arrowhead’s proprietary Targeted RNAi Molecule (TRiM) platform, Redemplo aims to restore triglyceride metabolism in a disease where conventional lipid-lowering agents are largely ineffective.

FCS is an ultra-rare disorder, estimated to affect between 1 and 13 people per million globally, and remains widely underdiagnosed. The condition is characterized by triglyceride levels that can reach orders of magnitude above the normal range — typically exceeding 10 mmol/L (880 mg/dL) — driven by loss-of-function mutations affecting lipoprotein lipase activity. The clinical consequences extend beyond lipid abnormalities: patients face a lifelong risk of acute and potentially fatal pancreatitis, alongside chronic abdominal pain, hepatic steatosis, and cognitive impairment. Standard lipid-lowering therapies, including fibrates and omega-3 fatty acids, do not adequately address the LPL-independent pathophysiology of FCS, and dietary fat restriction — often to less than 15 grams per day — remains the primary management strategy despite its limitations.

The CHMP opinion was supported by data from the Phase III PALISADE study (NCT05089084), a randomized, double-blind, placebo-controlled trial enrolling 75 adults with genetically confirmed or clinically diagnosed FCS across 39 sites in 18 countries. The trial’s primary endpoint was percent change from baseline in fasting triglycerides versus placebo at month 10. Patients receiving 25 mg plozasiran once every three months achieved a median 80% reduction in triglycerides from baseline, compared with a 17% reduction in the placebo group. The study also met all multiplicity-controlled key secondary endpoints, including significant reductions in apoC-III levels and a lower incidence of acute pancreatitis in the pooled plozasiran dose groups relative to placebo. Results from PALISADE were published in the New England Journal of Medicine and presented at the European Society of Cardiology Congress 2024 and the American Heart Association Scientific Sessions 2024. A subsequent 2025 analysis published in the Journal of Clinical Lipidology further characterized plozasiran’s effect on acute pancreatitis risk and potential quality-of-life benefit.

The most commonly reported adverse reactions in the trial were hyperglycemia (12.8%), headache (6.8%), nausea (4.7%), and injection site reaction (4.7%). Plozasiran is self-administered via subcutaneous injection once every three months.

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The EU regulatory landscape for FCS currently includes two antisense oligonucleotide therapies targeting apolipoprotein C-III (apoC-III) mRNA: Ionis Pharma’s Waylivra (volanesorsen), approved by the EMA in 2019 and restricted to patients with genetically confirmed FCS; and AstraZeneca’s Tryngolza (olezarsen), approved in 2025 for the same genetically defined population. Volanesorsen requires weekly subcutaneous administration and routine platelet monitoring due to the risk of thrombocytopenia—an issue that contributed to the US Food and Drug Administration declining its approval in the US.

Against this backdrop, the CHMP noted that plozasiran’s proposed indication does not require genetic confirmation of FCS, potentially extending treatment access to a broader population of adults with the condition. This distinction is clinically meaningful given the well-documented diagnostic challenges in FCS, where genetic confirmation is not always achievable and delays in diagnosis can exacerbate disease burden.

Arrowhead’s pipeline extends beyond FCS. Plozasiran is under Phase III investigation for severe hypertriglyceridemia in the SHASTA-3 (NCT06347003), SHASTA-4 (NCT06347016), and SHASTA-5 (NCT06880770) studies, and for hypertriglyceridemia more broadly in the MUIR-3 trial (NCT06347133). The US FDA granted plozasiran Breakthrough Therapy designation for severe hypertriglyceridemia in December 2025, indicating the molecule’s potential relevance across a wider spectrum of triglyceride-related disease than the rare FCS population alone.

The positive CHMP opinion positions Redemplo within a growing field of RNA-based therapeutics targeting metabolic disease, and adds to an expanding body of evidence that siRNA approaches can achieve durable, clinically meaningful effects through infrequent dosing. For the FCS community in Europe — a population that has had limited pharmacological options and, for clinically diagnosed patients, effectively none — the anticipated European Commission decision represents a potential shift in available care.