FDA approves Celcuity’s Revtorpyk as first dual PI3K/mTOR inhibitor for PIK3CA wild-type breast cancer

Minneapolis-based Celcuity Inc. (Nasdaq: CELC) has received US FDA approval for Revtorpyk (gedatolisib), making it the first and only therapy to inhibit all class I PI3K isoforms and both mTOR complexes to reach regulatory approval. The indication covers adult patients with HR+/HER2− locally advanced or metastatic breast cancer without a detected PIK3CA mutation, following progression on at least one line of endocrine therapy in the metastatic setting — a biomarker-defined population that has lacked targeted options within the PI3K/AKT/mTOR pathway.

Revtorpyk is approved in combination with fulvestrant, with or without palbociclib, administered intravenously. The drug inhibits all four class I PI3K isoforms (α, β, δ, γ) alongside mTORC1 and mTORC2, suppressing downstream AKT signaling while blocking the compensatory feedback reactivation that limits the efficacy of single-node inhibitors. This broad coverage of the PI3K/AKT/mTOR axis is the mechanistic rationale for its activity in PIK3CA wild-type tumors, where pathway activation occurs through mechanisms other than PIK3CA mutation.

The approval is supported by data from the Phase III VIKTORIA-1 trial, an open-label, global, randomized study in patients with locally advanced or metastatic HR+/HER2− breast cancer who had progressed on CDK4/6 therapy and an aromatase inhibitor. In the PIK3CA wild-type cohort, the Revtorpyk triplet (gedatolisib plus palbociclib and fulvestrant) demonstrated a median progression-free survival of 9.3 months versus 2.0 months with fulvestrant alone (HR=0.24; 95% CI: 0.17–0.35; p<0.0001), with an objective response rate of 32% compared to 1%. The doublet (gedatolisib plus fulvestrant) also showed statistically significant improvement, with a median PFS of 7.4 months versus 2.0 months (HR=0.33; p<0.0001).

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The approval positions Revtorpyk in a competitive landscape shaped by the PI3K pathway’s centrality to HR+/HER2− breast cancer resistance. AstraZeneca’s capivasertib (Truqap) plus fulvestrant is approved in the same post-endocrine therapy setting but is restricted to tumors harboring PIK3CA, AKT1, or PTEN alterations — leaving PIK3CA wild-type patients without AKT1 or PTEN alterations outside its label. Novartis’s everolimus (Afinitor) plus exemestane carries no biomarker restriction but targets only mTORC1, leaving compensatory pathway reactivation via mTORC2 intact. The competitive intensity in the PI3K space continues to grow, with Novartis recently committing up to USD 3 billion to acquire a next-generation PI3Kα inhibitor program.

Celcuity plans a commercial launch in late Q3 2026 and intends to file a supplemental NDA in Q3 2026 for the PIK3CA-mutant population, based on the mutant cohort of VIKTORIA-1. The ongoing Phase III VIKTORIA-2 trial is evaluating gedatolisib-based regimens in the first-line setting.


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