HutchMed’s Orpathys becomes first MET inhibitor approved for gastric cancer in China

HutchMed (China) Limited (Nasdaq: HCM) has received conditional NMPA approval for Orpathys (savolitinib) as the first selective MET inhibitor indicated for locally advanced or metastatic gastric cancer or gastroesophageal junction adenocarcinoma with MET amplification in China — addressing a molecularly defined patient population with limited post-second-line options. The approval, granted for patients who have failed at least two prior systemic treatments, marks the third approved indication for savolitinib in China and extends a precision oncology framework previously established in non-small cell lung cancer into gastrointestinal oncology.

Savolitinib is an oral, highly selective MET tyrosine kinase inhibitor that blocks aberrant activation of the MET receptor pathway driven by gene amplification, exon 14 skipping mutations, or protein overexpression. The drug is jointly developed by HutchMed and AstraZeneca, with AstraZeneca leading commercialization. The conditional approval reflects China’s regulatory pathway for therapies supported by single-arm Phase II data with objective response as the primary endpoint — the same route used for other HutchMed oncology assets progressing through the NMPA.

The approval is supported by a pivotal Phase II registration study (NCT04923932) conducted in MET-amplified gastric or gastroesophageal junction adenocarcinoma patients in China. The study met its primary endpoint of objective response rate as assessed by an Independent Review Committee per RECIST 1.1, reporting an IRC-assessed ORR of 32.3% (95% CI: 21.2%, 45.1%) as of the October 2025 data cutoff — exceeding the pre-specified efficacy threshold. Secondary endpoints included a disease control rate of 63.1%, median duration of response of 9.7 months, and median progression-free survival of 4.0 months. Results were published in Nature Medicine and presented at the ASCO Annual Meeting.

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MET amplification occurs in approximately 4–6% of gastric cancer patients, corresponding to an estimated 18,000 new cases annually in China. The pathway drives aggressive tumor behavior, and MET-amplified gastric cancer carries a poor prognosis relative to other molecular subtypes. Savolitinib selectively inhibits MET kinase activity, suppressing downstream signaling through the RAS-MAPK and PI3K-AKT pathways without the off-target kinase activity associated with earlier multi-kinase inhibitors such as crizotinib, which had been evaluated in MET-amplified gastroesophageal tumors in basket trial settings.

No approved MET-specific therapy for gastric cancer existed globally prior to this decision. The most direct pipeline competitor is glumetinib (SCC244), a selective MET inhibitor developed by Haihe Biopharma with an NDA submitted to the NMPA for MET-amplified gastric cancer — positioning savolitinib as the first approved agent in this molecular subtype but with a near-term competitive challenge in the same geography and biomarker population. More broadly, China’s gastric cancer precision oncology landscape has become increasingly active, with recent NMPA approvals across distinct biomarker-selected populations including Claudin18.2 and HER2, reflecting a broader shift toward molecularly stratified treatment in a disease historically managed with chemotherapy-based regimens.


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