Development

Cue’s anti-IgE antibody CUE-221 meets Phase II endpoints in chronic urticaria

Cue’s anti-IgE antibody CUE-221 meets Phase II endpoints in chronic urticaria

CUE-221 (UB-221), a dual-mechanism anti-IgE monoclonal antibody being developed by Boston-based Cue Biopharma (Nasdaq: CUE), met both its primary and key secondary endpoints in a Phase II study in chronic spontaneous urticaria (CSU), according to topline results released September 20. The data, generated in China by Genesis Life Sciences under an arrangement with Cue's China-based licensor Ascendant Health Limited, support progression to a Phase IIb/III registration-enabling study.

The randomized, double-blind, placebo- and active comparator-controlled trial enrolled 145 adults with moderate to severe CSU inadequately controlled despite H1 antihistamines. Patients received subcutaneous CUE-221 at 4 mg/kg, 2 mg/kg, or 1 mg/kg every four weeks (Q4W), placebo Q4W, or Novartis's Xolair (omalizumab) 300 mg Q4W. The primary endpoint — proportion of patients achieving a hive severity score of zero (HSS7=0) at week 12 — was met at all three dose levels, with rates of 54%, 53%, and 43% respectively versus 11% for placebo (p<0.005, p<0.005, p<0.05). The key secondary endpoint, complete response defined as UAS7=0 at week 12, reached statistical significance at the 4 mg/kg dose (46% versus 11% for placebo, p<0.05). Omalizumab achieved HSS7=0 in 41% of patients at week 12; statistical comparison against CUE-221 was not pre-specified.

CUE-221 (also known as UB-221) binds free IgE at epitopes distinct from those targeted by omalizumab, blocking its interaction with the high-affinity IgE receptor (FcεRI) on mast cells and basophils while preserving IgE's ability to engage the CD23 receptor on B cells — a mechanism the company says suppresses new IgE synthesis over time.

At week 22 — six weeks after the final dose — the 4 mg/kg group reached a peak HSS7=0 rate of 69%, compared with 41% for omalizumab. At week 28, twelve weeks after the last dose, 60% of patients in the 4 mg/kg arm maintained complete resolution of hives versus 24% in the omalizumab arm; a post hoc analysis reported this difference as statistically significant (delta = 36%, p<0.05). The post hoc nature of the week-28 comparison limits its evidentiary weight, and the omalizumab arm was not powered for statistical testing. Complete PK and IgE analyses from the 36-week study have not yet been disclosed.

CUE-221 demonstrated a favorable safety profile with no treatment-related serious adverse events and no hypersensitivity reactions including anaphylaxis. Injection site reactions were infrequent, with only one exceeding grade 1.

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The CSU treatment landscape has expanded substantially since 2024. Novartis's Xolair has been the standard second-line biologic since 2014. Sanofi and Regeneron's Dupixent (dupilumab), which blocks IL-4/IL-13 signaling rather than IgE, received FDA approval for antihistamine-refractory CSU in April 2025. Novartis's Rhapsido (remibrutinib), an oral BTK inhibitor that suppresses mast cell degranulation downstream of FcεRI, was approved in September 2025 — making it the only oral approved therapy in this setting. CUE-221 would enter a market that now includes three distinct approved mechanisms.

Cue in-licensed ex-Greater China rights to CUE-221 from Ascendant Health Sciences in April 2026 for USD 15 million upfront and up to USD 676.5 million in additional milestones. Development rights in mainland China, Hong Kong, Macau, and Taiwan remain with Genesis Life Sciences, also known as Yangzhou Shizhiyuan Biotechnology, which conducted the Phase II CSU study. Shanghai-listed Shenlian Biomedical acquired control of Genesis in March 2026 through a transaction that gave Shenlian and an acting-in-concert vehicle a combined 51% stake, marking Shenlian's expansion from animal health into human innovative drugs. Genesis and Ascendant are related companies, with Ascendant holding the ex-Greater China rights subsequently licensed to Cue.


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