Development

Tagrisso plus Orpathys extends OS in MET-resistant EGFR-mutated lung cancer

The SAFFRON Phase III trial has delivered the first global Phase III evidence that a targeted oral combination can improve both progression-free and overall...

Tagrisso plus Orpathys extends OS in MET-resistant EGFR-mutated lung cancer

The SAFFRON Phase III trial has shown that an oral targeted combination can improve both progression-free and overall survival in patients with EGFR-mutant non-small cell lung cancer (NSCLC) and MET-driven resistance to osimertinib, according to AstraZeneca (LSE/STO/NYSE: AZN). The result could establish a new biomarker-directed treatment option for patients who progress on Tagrisso (osimertinib).

The global trial tested savolitinib (Orpathys), a selective MET tyrosine kinase inhibitor (TKI) co-developed by AstraZeneca and HUTCHMED (China) Limited (Nasdaq/AIM: HCM; HKEX: 13), in combination with osimertinib against platinum-based doublet chemotherapy. SAFFRON enrolled 345 patients with EGFR-mutant locally advanced or metastatic NSCLC with high MET overexpression or amplification whose disease had progressed on first- or second-line osimertinib.

The combination met the primary endpoint of progression-free survival (PFS) and key secondary endpoint of overall survival (OS), producing statistically significant and clinically meaningful improvements over chemotherapy, the companies said. No hazard ratios or median PFS or OS figures were disclosed in the top-line announcement, with full results due at a forthcoming medical meeting. The safety profile was consistent with the established profiles of the two drugs, with no new safety findings reported.

MET amplification and overexpression are important mechanisms of acquired resistance to EGFR TKIs. Savolitinib inhibits aberrant MET signaling while continued osimertinib treatment maintains EGFR suppression, allowing the combination to target both the original oncogenic driver and a major pathway through which tumors can escape EGFR inhibition. AstraZeneca estimates that high-level MET overexpression or amplification occurs in approximately 34% of tumors following progression on third-generation EGFR TKIs.

Osimertinib is already widely approved across EGFR-mutant NSCLC, while the savolitinib-osimertinib combination is approved in China for patients with MET-amplified disease following EGFR-TKI treatment. That approval was supported by the SACHI Phase III trial, which reported median investigator-assessed PFS of 8.2 months with the combination versus 4.5 months with chemotherapy. The combination has also received temporary authorization in Switzerland based on Phase II SAVANNAH data. SAFFRON now provides global Phase III evidence that could support regulatory filings in the US, EU, and other markets.

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Competition in the post-osimertinib setting includes Johnson & Johnson's Rybrevant (amivantamab-vmjw), an EGFR-MET bispecific antibody approved by the US FDA in September 2024 with carboplatin and pemetrexed following EGFR-TKI progression. Unlike SAFFRON, that indication covers the broader post-EGFR-TKI population and does not require selection for MET-driven resistance. AstraZeneca and Daiichi Sankyo's TROP2-directed antibody-drug conjugate Datroway (datopotamab deruxtecan) is also approved for EGFR-mutant NSCLC following prior EGFR-directed therapy and platinum-based chemotherapy.

Savolitinib plus osimertinib could therefore become the first globally available all-oral, biomarker-directed regimen specifically for MET-driven resistance following osimertinib. The Phase III OS benefit is particularly important in a setting where treatment development has increasingly focused on overcoming molecular mechanisms of acquired resistance rather than moving patients directly to non-selective chemotherapy.

AstraZeneca and HUTCHMED plan to submit the SAFFRON results to regulatory authorities globally, with detailed findings to be presented at a forthcoming medical meeting.


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