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Merck strikes USD 2.13bn deal for China-origin preclinical KRAS G12D inhibitor

Merck strikes USD 2.13bn deal for China-origin preclinical KRAS G12D inhibitor

Merck (NYSE: MRK) has agreed to pay USD 400 million upfront to exclusively license SPR2015, a preclinical oral KRAS G12D-targeted RAS(ON) inhibitor from China-based SciBrunch Therapeutics. The deal could reach USD 2.13 billion including development, commercialization, and other milestone payments across multiple indications.

Why it matters: The USD 400 million upfront payment is unusually large for a preclinical asset and gives Merck an entry into the competitive race to inhibit active KRAS G12D, one of the most common oncogenic RAS mutations. Revolution Medicines has already advanced a competing RAS(ON) KRAS G12D inhibitor into clinical development.

Under the agreement, SciBrunch granted Merck exclusive worldwide rights to develop, manufacture, and commercialize SPR2015. The transaction has closed, and Merck expects to record a USD 400 million pre-tax charge in Q3 2026.

SPR2015 is an oral molecular glue designed to inhibit the active, or ON, state of KRAS G12D by forming a ternary complex with cyclophilin A and the mutant KRAS protein. Preclinical data presented at AACR 2026 showed selective activity in KRAS G12D-mutant cancer models, including pancreatic, lung, and colorectal tumors. The program has not yet entered clinical testing.

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SciBrunch was founded in late 2024 by Tao Hu and Yang Zhang, both with backgrounds in small-molecule drug discovery, and had raised approximately USD 65 million across two financing rounds before the Merck deal. Merck’s USD 400 million upfront payment is therefore more than six times the company’s disclosed private financing to date.

The closest clinical benchmark is zoldonrasib (RMC-9805), Revolution Medicines’ oral RAS(ON) KRAS G12D-selective inhibitor. Zoldonrasib also uses a cyclophilin A-dependent tri-complex mechanism and is currently under the US FDA's approval review for metastatic pancreatic ductal adenocarcinoma.


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