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BeOne and Revolution Medicines partner on RAS(ON) inhibitor combinations in Asia

BeOne and Revolution Medicines partner on RAS(ON) inhibitor combinations in Asia

BeOne Medicines Ltd. (Nasdaq: ONC; HKEX: 06160; SSE: 688235) and Revolution Medicines, Inc. (Nasdaq: RVMD) announced a two-part arrangement covering both clinical combination studies and regional commercialization rights for Revolution Medicines' four clinical-stage RAS(ON) inhibitors in select Asian markets. Notably, the deal will also see BeOne fund and conduct a global registrational Phase III study for one of the four RAS(ON) inhibitors, with the specific asset not identified in the announcement.

Under the deal terms, BeOne takes exclusive development and commercialization rights — or commercialization-only rights, depending on the market — to daraxonrasib, zoldonrasib, elironrasib, and RMC-5127 across those territories. Revolution is eligible to receive development and sales milestone payments plus tiered royalties on net sales within the licensed territory, with specifics not disclosed. The US firm explicitly retains all rights outside the licensed region, including Japan and South Korea.

The four Revolution assets span distinct RAS mutant variants: daraxonrasib is a multi-selective RAS(ON) inhibitor, zoldonrasib targets KRAS G12D, elironrasib targets KRAS G12C, and RMC-5127 targets KRAS G12V. All four inhibit the active, GTP-bound state of oncogenic RAS proteins, differentiating them from approved KRAS G12C inhibitors that target the inactive, GDP-bound state. The clinical collaboration will evaluate combinations of these four agents with two BeOne pipeline assets: BGB-58067, an MTA-cooperative PRMT5 inhibitor, and BG-T187, an EGFR x MET x MET trispecific antibody. Initial planned studies will pair BGB-58067 and BG-T187 with either daraxonrasib or zoldonrasib.

The deal gives BeOne access to a RAS(ON) portfolio that has gained substantial clinical validation, led by daraxonrasib in pancreatic ductal adenocarcinoma (PDAC). In the 500-patient Phase III RASolute 302 trial in previously treated metastatic PDAC, daraxonrasib reduced the risk of death by 60% versus investigator's choice of chemotherapy, with median overall survival of 13.2 months versus 6.7 months; the results supported an NDA accepted for US FDA review in July 2026. Revolution has also reported encouraging early activity with KRAS G12D-selective zoldonrasib, including a 52% confirmed objective response rate in previously treated KRAS G12D-mutant NSCLC, alongside combination activity in pancreatic cancer. Together, the portfolio gives BeOne exposure to both mutation-selective and multi-RAS targeting strategies rather than tying the collaboration to the success of a single molecular subtype.

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The clinical collaboration also provides a rationale for combining RAS inhibition with complementary mechanisms from BeOne's oncology pipeline. BGB-58067 is an MTA-cooperative PRMT5 inhibitor, a drug class being developed particularly in MTAP-deleted cancers, while BG-T187 simultaneously targets EGFR and MET, two receptor tyrosine kinase pathways implicated in tumor growth and adaptive resistance to targeted therapy. Initial studies pairing these agents with daraxonrasib or zoldonrasib could test whether simultaneous blockade of RAS and complementary signaling or survival pathways can deepen responses or delay resistance beyond what is achievable with RAS(ON) inhibition alone.

Notably, BeOne's recent Q2 2026 earnings call revealed that the company is already building a broader internal RAS strategy. BeOne disclosed plans to move a CNS-penetrant RAS(ON) inhibitor into clinical development before year-end, alongside a KRAS-targeting degrader and a RAS(ON)-based ADC concept. Adding Revolution's clinically advanced portfolio gives BeOne exposure to multiple RAS mutations and stages of development while retaining differentiated internal approaches to the target, making RAS-directed therapy an increasingly significant component of its solid tumor strategy.


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